Research Article

Open Access
SLC38A9 is directly involved in Tat-induced endolysosome dysfunction and senescence in astrocytes
Neda Rezagholizadeh, Gaurav Datta, Wendie A Hasler, Erica C Nguon, Elise V Smokey, Nabab Khan, View ORCID ProfileXuesong Chen Correspondence email
Neda Rezagholizadeh
Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA
Roles: Conceptualization, Data curation, Formal analysis, Validation, Investigation, Methodology, Writing—original draft
Gaurav Datta
Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA
Roles: Formal analysis, Investigation, Methodology
Wendie A Hasler
Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA
Roles: Formal analysis, Investigation
Erica C Nguon
Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA
Roles: Investigation
Elise V Smokey
Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA
Roles: Investigation
Nabab Khan
Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA
Roles: Formal analysis, Investigation, Methodology
Xuesong Chen
Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA
Roles: Conceptualization, Data curation, Funding acquisition, Validation, Writing—review and editing
Published 5 May 2025. DOI: 10.26508/lsa.202503231

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SLC38A9 in Tat-induced senescence
Neda Rezagholizadeh, Gaurav Datta, Wendie A Hasler, Erica C Nguon, Elise V Smokey, Nabab Khan, Xuesong Chen
Life Science Alliance May 2025, 8 (7) e202503231; DOI: 10.26508/lsa.202503231
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Volume 8, No. 7
July 2025
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