miR-486 is essential for muscle function and suppresses a dystrophic transcriptome

Adrienne Samani; Rylie M Hightower; Andrea L Reid; Katherine G English; Michael A Lopez; J Scott Doyle; Michael J Conklin; David A Schneider; Marcas M Bamman; Jeffrey J Widrick; David K Crossman; Min Xie; David Jee; Eric C Lai; Matthew S Alexander

doi:10.26508/lsa.202101215

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miR-486 is essential for muscle function and suppresses a dystrophic transcriptome

Adrienne Samani, Rylie M Hightower, Andrea L Reid, View ORCID ProfileKatherine G English, View ORCID ProfileMichael A Lopez, J Scott Doyle, Michael J Conklin, View ORCID ProfileDavid A Schneider, Marcas M Bamman, View ORCID ProfileJeffrey J Widrick, View ORCID ProfileDavid K Crossman, Min Xie, David Jee, View ORCID ProfileEric C Lai, View ORCID ProfileMatthew S Alexander Correspondence email

Adrienne Samani

1Department of Pediatrics, Division of Neurology at Children’s of Alabama and the University of Alabama at Birmingham, Birmingham, AL, USA

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Rylie M Hightower

1Department of Pediatrics, Division of Neurology at Children’s of Alabama and the University of Alabama at Birmingham, Birmingham, AL, USA

2University of Alabama at Birmingham Center for Exercise Medicine (UCEM), Birmingham, AL, USA

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Andrea L Reid

1Department of Pediatrics, Division of Neurology at Children’s of Alabama and the University of Alabama at Birmingham, Birmingham, AL, USA

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Katherine G English

1Department of Pediatrics, Division of Neurology at Children’s of Alabama and the University of Alabama at Birmingham, Birmingham, AL, USA

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ORCID record for Katherine G English

Michael A Lopez

1Department of Pediatrics, Division of Neurology at Children’s of Alabama and the University of Alabama at Birmingham, Birmingham, AL, USA

2University of Alabama at Birmingham Center for Exercise Medicine (UCEM), Birmingham, AL, USA

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J Scott Doyle

3Department of Orthopedic Surgery, at the University of Alabama at Birmingham, Birmingham, AL, USA

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Michael J Conklin

3Department of Orthopedic Surgery, at the University of Alabama at Birmingham, Birmingham, AL, USA

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David A Schneider

4Department of Biochemistry and Molecular Genetics at the University of Alabama at Birmingham, Birmingham, AL, USA

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Marcas M Bamman

2University of Alabama at Birmingham Center for Exercise Medicine (UCEM), Birmingham, AL, USA

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Jeffrey J Widrick

5Division of Genetics and Genomics at Boston Children’s Hospital, Boston, MA, USA

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David K Crossman

6Department of Genetics, University of Alabama at Birmingham, Birmingham, AL, USA

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Min Xie

7Division of Cardiovascular Disease, Department of Medicine, University of Alabama at Birmingham, School of Medicine, Birmingham, AL, USA

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David Jee

8Developmental Biology Program, Sloan Kettering Institute, New York, NY, USA

9Weill Graduate School of Medical Sciences, Cornell University, New York, NY, USA

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Eric C Lai

8Developmental Biology Program, Sloan Kettering Institute, New York, NY, USA

9Weill Graduate School of Medical Sciences, Cornell University, New York, NY, USA

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Matthew S Alexander

1Department of Pediatrics, Division of Neurology at Children’s of Alabama and the University of Alabama at Birmingham, Birmingham, AL, USA

2University of Alabama at Birmingham Center for Exercise Medicine (UCEM), Birmingham, AL, USA

6Department of Genetics, University of Alabama at Birmingham, Birmingham, AL, USA

10UAB Civitan International Research Center (CIRC), at the University of Alabama at Birmingham, Birmingham, AL, USA

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ORCID record for Matthew S Alexander
For correspondence: matthewalexander@uabmc.edu

Published 5 May 2022. DOI: 10.26508/lsa.202101215

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miR-486 regulates normal and dystrophic muscle function

Adrienne Samani, Rylie M Hightower, Andrea L Reid, Katherine G English, Michael A Lopez, J Scott Doyle, Michael J Conklin, David A Schneider, Marcas M Bamman, Jeffrey J Widrick, David K Crossman, Min Xie, David Jee, Eric C Lai, Matthew S Alexander

Life Science Alliance May 2022, 5 (9) e202101215; DOI: 10.26508/lsa.202101215

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miR-486 regulates normal and dystrophic muscle function

Life Science Alliance May 2022, 5 (9) e202101215; DOI: 10.26508/lsa.202101215

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September 2022

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