Research Article
Open Access
Septins tune lipid kinase activity and PI(4,5)P2 turnover during G-protein–coupled PLC signalling in vivo
View ORCID ProfileAastha Kumari, View ORCID ProfileAvishek Ghosh, View ORCID ProfileSourav Kolay, View ORCID ProfilePadinjat Raghu Correspondence email
Aastha Kumari
1National Centre for Biological Sciences, TIFR-GKVK Campus, Bengaluru, India
Roles: Resources, Formal analysis, Validation, Investigation, Methodology, Writing—original draft, review, and editing
Avishek Ghosh
1National Centre for Biological Sciences, TIFR-GKVK Campus, Bengaluru, India
3Department of Surgery, Vascular Biology Program, Boston Children’s Hospital, Harvard Medical School, Boston, MA, USA
Roles: Formal analysis, Methodology, Writing—original draft, review, and editing
Sourav Kolay
1National Centre for Biological Sciences, TIFR-GKVK Campus, Bengaluru, India
2UT Southwestern Medical Center, Dallas, TX, USA
Roles: Formal analysis
Padinjat Raghu
1National Centre for Biological Sciences, TIFR-GKVK Campus, Bengaluru, India
Roles: Conceptualization, Supervision, Project administration, Writing—original draft, review, and editing
Published 11 March 2022. DOI: 10.26508/lsa.202101293
Regulation of PLC signalling by septins
Aastha Kumari, Avishek Ghosh, Sourav Kolay, Padinjat Raghu
Life Science Alliance Mar 2022, 5 (6) e202101293; DOI: 10.26508/lsa.202101293
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Volume 5, No. 6
June 2022
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