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- D614G reshapes allosteric networks and opening mechanisms of SARS-CoV-2 spikes
- Structural basis of TMPRSS11D specificity and autocleavage activation
- Resolving the Structure of a Guanine Quadruplex in TMPRSS2 Messenger RNA by Circular Dichroism and Molecular Modeling
- ACE2 from Pipistrellus abramus bats is a receptor for HKU5 coronaviruses
- In-silico docking platform with serine protease inhibitor (SERPIN) structures identifies host cysteine protease targets with significance for SARS-CoV-2
- Cholesterol and COVID-19--therapeutic opportunities at the host/virus interface during cell entry
- Efficacy of Host Cell Serine Protease Inhibitor MM3122 against SARS-CoV-2 for Treatment and Prevention of COVID-19
- TMPRSS2 activation of Omicron lineage Spike glycoproteins is regulated by TMPRSS2 cleavage of ACE2
- ACE2-EGFR-MAPK signaling contributes to SARS-CoV-2 infection
- Expression of TMPRSS2 is up-regulated by bacterial flagellin, LPS, and Pam3Cys in human airway cells
- Furin as a therapeutic target in cystic fibrosis airways disease
- Determinants and Mechanisms of the Low Fusogenicity and Endosomal Entry of Omicron Subvariants
- Rapalogs downmodulate intrinsic immunity and promote cell entry of SARS-CoV-2
- The diversity of the glycan shield of sarbecoviruses closely related to SARS-CoV-2
- Evolution of host protease interactions among SARS-CoV-2 variants of concern and related coronaviruses
- Tracking infectious entry routes of SARS-CoV-2
- ACE2-independent SARS-CoV-2 infection and mouse adaption emerge after passage in cells expressing human and mouse ACE2
- SARS-CoV-2 Omicron spike H655Y mutation is responsible for enhancement of the endosomal entry pathway and reduction of cell surface entry pathways
- A Suite of TMPRSS2 Assays for Screening Drug Repurposing Candidates as Potential Treatments of COVID-19
- Effect of SARS-CoV-2 spike mutations on its activation by TMPRSS2 and TMPRSS13
- IL4I1 binds to TMPRSS13 and competes with SARS-Cov2 Spike
- Metalloproteinase-dependent and TMPRSS2-independnt cell surface entry pathway of SARS-CoV-2 requires the furin-cleavage site and the S2 domain of spike protein
- A novel class of TMPRSS2 inhibitors potently block SARS-CoV-2 and MERS-CoV viral entry and protect human epithelial lung cells
- A diabetic milieu increases cellular susceptibility to SARS-CoV-2 infections in engineered human kidney organoids and diabetic patients
- Effect of natural mutations of SARS-CoV-2 on spike structure, conformation, and antigenicity
- A method for the generation of pseudotyped virus particles bearing SARS coronavirus spike protein in high yields
- FXa cleaves the SARS-CoV-2 spike protein and blocks cell entry to protect against infection with inferior effects in B.1.1.7 variant
- The PRRA Insert at the S1/S2 Site Modulates Cellular Tropism of SARS-CoV-2 and ACE2 Usage by the Closely Related Bat RaTG13
- The Furin-S2' Site in Avian Coronavirus Plays a Key Role in Central Nervous System Damage Progression
- On the Origin of SARS-CoV-2: Did Cell Culture Experiments Lead to Increased Virulence of the Progenitor Virus for Humans?
- SARS-CoV-2 Causes Severe Epithelial Inflammation and Barrier Dysfunction
- Large fluctuations of the fusion intermediate help SARS-CoV-2 capture host cell membranes
- Term Human Placental Trophoblasts Express SARS-CoV-2 Entry Factors ACE2, TMPRSS2, and Furin
- Crystal structure of inhibitor-bound human MSPL that can activate high pathogenic avian influenza
- Structural basis for GTP-induced dimerization and antiviral function of guanylate-binding proteins
- Coagulation factors directly cleave SARS-CoV-2 spike and enhance viral entry
- Crystal structure of inhibitor-bound human MSPL that can activate high pathogenic avian influenza
- SPINT2 controls SARS-CoV-2 viral infection and is associated to disease severity
- Spike Protein of SARS-CoV-2 Activates Macrophages and Contributes to Induction of Acute Lung Inflammations in Mice
- A monoclonal antibody against staphylococcal enterotoxin B superantigen inhibits SARS-CoV-2 entry in vitro
- The rocaglate CR-31-B (-) inhibits SARS-CoV-2 replication at non-cytotoxic, low nanomolar concentrations in vitro and ex vivo
- The SARS-CoV-2 and other human coronavirus spike proteins are fine-tuned towards temperature and proteases of the human airways
- Single-cell analyses reveal SARS-CoV-2 interference with intrinsic immune response in the human gut
- Safe and effective two-in-one replicon-and-VLP minispike vaccine for COVID-19
- A Tethered Ligand Assay to Probe the SARS-CoV-2 ACE2 Interaction under Constant Force