Research Article


Open Access
Caspase-1 cleaves Bid to release mitochondrial SMAC and drive secondary necrosis in the absence of GSDMD
Rosalie Heilig, Marisa Dilucca, View ORCID ProfileDave Boucher, View ORCID ProfileKaiwen W Chen, Dora Hancz, View ORCID ProfileBenjamin Demarco, Kateryna Shkarina, View ORCID ProfilePetr Broz Correspondence email
Rosalie Heilig
Department of Biochemistry, University of Lausanne, Epalinges, Switzerland
Marisa Dilucca
Department of Biochemistry, University of Lausanne, Epalinges, Switzerland
Dave Boucher
Department of Biochemistry, University of Lausanne, Epalinges, Switzerland
Kaiwen W Chen
Department of Biochemistry, University of Lausanne, Epalinges, Switzerland
Dora Hancz
Department of Biochemistry, University of Lausanne, Epalinges, Switzerland
Benjamin Demarco
Department of Biochemistry, University of Lausanne, Epalinges, Switzerland
Kateryna Shkarina
Department of Biochemistry, University of Lausanne, Epalinges, Switzerland
Petr Broz
Department of Biochemistry, University of Lausanne, Epalinges, Switzerland
Published 28 April 2020. DOI: 10.26508/lsa.202000735
GSDMD-independent cell death requires casp1-driven Bid truncation
Rosalie Heilig, Marisa Dilucca, Dave Boucher, Kaiwen W Chen, Dora Hancz, Benjamin Demarco, Kateryna Shkarina, Petr Broz
Life Science Alliance Apr 2020, 3 (6) e202000735; DOI: 10.26508/lsa.202000735
In this Issue
Volume 3, No. 6
June 2020
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