Research Article
Open Access
An N-terminal–truncated isoform of FAM134B (FAM134B-2) regulates starvation-induced hepatic selective ER-phagy
Shohei Kohno, Yuji Shiozaki, Audrey L Keenan, Shinobu Miyazaki-Anzai, View ORCID ProfileMakoto Miyazaki Correspondence email
Shohei Kohno
Division of Renal Diseases and Hypertension, Department of Medicine, University of Colorado Denver, Aurora, CO, USA
Yuji Shiozaki
Division of Renal Diseases and Hypertension, Department of Medicine, University of Colorado Denver, Aurora, CO, USA
Audrey L Keenan
Division of Renal Diseases and Hypertension, Department of Medicine, University of Colorado Denver, Aurora, CO, USA
Shinobu Miyazaki-Anzai
Division of Renal Diseases and Hypertension, Department of Medicine, University of Colorado Denver, Aurora, CO, USA
Makoto Miyazaki
Division of Renal Diseases and Hypertension, Department of Medicine, University of Colorado Denver, Aurora, CO, USA
Published 17 May 2019. DOI: 10.26508/lsa.201900340
Statistics from Altmetric.com
Article usage
Starvation-induced hepatic ER-phagy
Shohei Kohno, Yuji Shiozaki, Audrey L Keenan, Shinobu Miyazaki-Anzai, Makoto Miyazaki
Life Science Alliance May 2019, 2 (3) e201900340; DOI: 10.26508/lsa.201900340
In this Issue
Volume 2, No. 3
June 2019
Advertisement
Jump to section
Related Articles
- No related articles found.
Cited By...
- The ER-phagy receptor FAM134B is targeted by Salmonella Typhimurium to promote infection
- Mitochondrial Sodium/Calcium Exchanger (NCLX) Regulates Basal and Starvation-Induced Autophagy Through Calcium Signaling
- ER-Phagy: Quality and Quantity Control of the Endoplasmic Reticulum by Autophagy
- Targeted Disruption of a Proximal Tubule-Specific TMEM174 Gene in Mice Causes Hyperphosphatemia and Vascular Calcification