Research Article
Open Access
Opposing functions of F-BAR proteins in neuronal membrane protrusion, tubule formation, and neurite outgrowth
Kendra L Taylor, Russell J Taylor, Karl E Richters, Brandon Huynh, Justin Carrington, Maeve E McDermott, Rebecca L Wilson, View ORCID ProfileErik W Dent Correspondence email
Kendra L Taylor
1University of Wisconsin-Madison, Neuroscience Training Program, Madison, WI, USA
Russell J Taylor
1University of Wisconsin-Madison, Neuroscience Training Program, Madison, WI, USA
Karl E Richters
2University of Wisconsin-Madison, Department of Neuroscience, Madison, WI, USA
Brandon Huynh
2University of Wisconsin-Madison, Department of Neuroscience, Madison, WI, USA
Justin Carrington
2University of Wisconsin-Madison, Department of Neuroscience, Madison, WI, USA
Maeve E McDermott
2University of Wisconsin-Madison, Department of Neuroscience, Madison, WI, USA
Rebecca L Wilson
2University of Wisconsin-Madison, Department of Neuroscience, Madison, WI, USA
Erik W Dent
2University of Wisconsin-Madison, Department of Neuroscience, Madison, WI, USA
Published 3 June 2019. DOI: 10.26508/lsa.201800288
Statistics from Altmetric.com
Article usage
CIP4 protrusion and FBP17 tubulation
Kendra L Taylor, Russell J Taylor, Karl E Richters, Brandon Huynh, Justin Carrington, Maeve E McDermott, Rebecca L Wilson, Erik W Dent
Life Science Alliance Jun 2019, 2 (3) e201800288; DOI: 10.26508/lsa.201800288
In this Issue
Volume 2, No. 3
June 2019
Advertisement
Jump to section
Subjects
Related Articles
- No related articles found.
Cited By...
- Self-assembly of CIP4 drives actin-mediated asymmetric pit-closing in clathrin-mediated endocytosis
- Self-assembly of CIP4 drives actin-mediated asymmetric pit-closing in clathrin-mediated endocytosis
- Filopodial protrusion driven by density-dependent Ena-TOCA-1 interactions
- Self-assembly of CIP4 drives actin-mediated asymmetric pit-closing in clathrin-mediated endocytosis